Abilities
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Bacterial Pneumonia
Pathogen damages pulmonary lining, allowing lethal secondary bacterial infections. Antibiotics will reduce impact
Cold Resistance 1
Pathogen evolves to withstand cold temperatures and climates
Cold Resistance 2
Lower intracellular water volume prevents freezing. Increased effectiveness in cold climates
Drug Resistance 1
Pathogen becomes resistant to class 1 and 2 antivirals. Increase effectiveness in wealthy countries
Drug Resistance 2
Pathogen develops resistance to class 3 and 4 antivirals. Increase effectiveness in wealthy countries
Environmental Hardening
Pathogen develops hardened coating - becoming highly weather resistant and extremely comfortable in both hot and cold climates
Genetic Hardening 1
Pathogen becomes harder to analyse in the lab. Decreases future research speed
Genetic Hardening 2
The pathogen does not reproduce in lab conditions. Decreases future research speed
Genetic ReShuffle 1
Pathogen DNA strands reassembled. More work needed to develop a cure
Genetic ReShuffle 2
A new strain of the pathogen now exists, increasing the work needed to develop a cure
Genetic ReShuffle 3
Multiple strains of the pathogen now exist, increasing the work needed to develop a cure
Heat Resistance 1
Pathogen evolves to withstand hot temperatures and climates
Heat Resistance 2
Pathogen avoids cellular breakdown in high temperatures. Increased effectiveness in hot climates
Historic Reassortment
Gives ability to access the genetic sequence expressed in the 1918 flu. Highly lethal strain
Viral Instability 1
DNA becomes more unstable, increasing random mutations
Viral Instability 2
DNA occasionally fractures, increasing random mutations
Viral Instability 3
DNA becomes highly erratic and fragmented, increasing random mutations